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Solved: How to update a shelf drawing, one shelf with several volumes?
Some of them are still not visible in 3D group preferences:
You have AutoCAD Civil 3D 2016 installed, therefore your active Civil 3D toolkit is 2015.
To make the tool buttons visible in the top of your drawing go to:
View > Toolbars > 3D
Make sure you see “2018” and “3D Commands” and on “top of the Autodesk 3D Command and Feature list”
Induction of cyclooxygenase-2 in response to tumor necrosis factor-alpha involves an activation of mitogen-activated protein kinases in mouse peritoneal macrophages.
An earlier work showed that the expression of inducible cyclooxygenase-2 (COX-2) can be induced by tumor necrosis factor (TNF)-alpha in mouse peritoneal macrophages. In this work, we show that different modes of TNF-alpha induction can be associated with distinct kinase activation. TNF-alpha-induced COX-2 expression was not inhibited by cycloheximide (CHX) in the presence of actinomycin D, suggesting that TNF-alpha does not require new protein synthesis. By contrast, CHX pretreatment inhibited the TNF-alpha-induced phosphorylation of mitogen-activated protein (MAP) kinases but not that of p44/42 MAP kinase. The inhibitory effect of CHX on MAP kinase phosphorylation in the presence of TNF-alpha was not abolished by the addition of exogenous protein synthesis, indicating that TNF-alpha can bypass the requirement for new protein synthesis during the signaling mechanism. To further support the existence of an alternative route of activation of MAP kinase signaling, the contribution of phosphatidylinositol 3-kinase (PI3-kinase) was tested. It was found that the TNF-alpha-induced phosphorylation of MAP kinases was abolished by pretreatment of peritoneal macrophages with wortmannin, a specific PI3-kinase inhibitor. The incubation of cells with TNF-alpha or LPS in the presence of the MAP kinase kinase (MEK) inhibitor PD98059 did not affect the induction of COX-2 promoter activity. In contrast
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